Table Legends
Table 1. Characteristics of study subjects. EC, Endemic control; LTBi, latent TB; DM, diabetes mellitus; DM-LTBi, latently infected Diabetics; TB, tuberculosis; RBS, random blood sugar.
Table 2. Differential pro-inflammatory cytokine secretion between healthy controls, diabetics and LTBi cases. EC, Endemic control; LTBi, latent TB; DM, diabetes mellitus; DM-LTBi, latently infected Diabetics; TB, tuberculosis. Supernatants were collected and tested for IFN-γ, IL-12p70, IL-2, TNF-α, GM-CSF, and IL-1β by Th1 Th2 11 plex procarta plex assay (Thermofischer, USA). Data is depicted as median with IQR. Kruskal Wallis test was run between the groups to see the difference in secretion levels and P values are included in the table. Individual groups were also compared using Mann–Whitney U-test. *Denotes a significant difference (≤0.05) as compared with DM-LTBi; # as compared with EC; NS, not significantly different, p>0.05.
Table 3. Differential type 2 cytokine secretion between healthy controls, diabetics and LTBi cases. EC, Endemic control; LTBi, latent TB; DM, diabetes mellitus; DM-LTBi, latently infected diabetics; TB, tuberculosis. Supernatants were collected and tested for IL-13, IL-4, IL-5, IL-6 and IL-18 by Th1 Th2 11 plex procarta plex assay (Thermofischer, USA). Data is depicted as median with IQR. Kruskal Wallis test was run between the groups to see the difference in secretion levels and P values are included in the table. Individual groups were also compared using Mann–Whitney U-test. *Denotes a significant difference (≤0.05) as compared with DM-LTBi; NS, not significantly different, p>0.05.