Introduction:

Thoracic outlet syndrome (TOS) is a constellation of symptoms in the shoulder and upper extremity that may present as pain, dysesthesia, weakness, edema, and pallor resulting from total or partial compression of the neurovascular bundle exiting the space between the first rib and the clavicle, known as the thoracic outlet. TOS is classified into three subgroups according to its pathophysiology: neurogenic TOS (nTOS), arterial TOS (aTOS), and venous TOS (vTOS). Among them, nTOS accounts for more than 90% of the cases, followed by vTOS accounting for 3-5%, and aTOS accounting for 1-2% [1]. Venous TOS, an uncommon subtype, occurs as a result of extrinsic compression and subsequent stenosis of the subclavian vein. It is a predominantly unilateral and is often associated with repetitive upper extremity motion. According to previous reports, anomalous anatomy, repetitive movement-induced swelling with microhemorrhage and subsequent fibrosis, compressive soft tissue masses, deep-venous thrombosis can be initiators of vTOS [2-5]. If the vTOS is caused by a spontaneous deep vein thrombosis (DVT), it is referred to as Paget-von Schröetter Syndrome.