CONCLUSION
Our patient was newly diagnosed with anti-GBM disease and presented with the unique pathophysiology of hypertensive emergency and subsequent encephalopathy in the form of seizures. He was successfully stabilized and managed with strict blood pressure and seizure control. Our report brings attention to the complications that may arise with anti-GBM disease. Our patient has not had any recurrence of seizures implying that the cause of his seizures was most likely related to his hypertensive crises and that adequate BP control can prevent seizures in patients with anti-GBM disease.