CONCLUSION
Our patient was newly diagnosed with anti-GBM disease and presented with
the unique pathophysiology of hypertensive emergency and subsequent
encephalopathy in the form of seizures. He was successfully stabilized
and managed with strict blood pressure and seizure control. Our report
brings attention to the complications that may arise with anti-GBM
disease. Our patient has not had any recurrence of seizures implying
that the cause of his seizures was most likely related to his
hypertensive crises and that adequate BP control can prevent seizures in
patients with anti-GBM disease.