Introduction
The incidence of coarctation of aorta (CoA) is 4 in 10,000 live births, and accounts for 5% to 8% of congenital heart diseases [1]. Patent ductus arteriosus, ventricular septal defect, bicuspid aortic valve, cerebral aneurysms of the circle of Willis and other congenital heart diseases (transposition of the great vessels, Tausig-Bing anomaly, hypoplastic left heart syndrome etc.) may accompany CoA [2,3]. In untreated cases, coronary disease, aortic dissection and rupture, cerebrovascular accident, or bacterial endocarditis may develop as complications, while the survival of these cases is approximately 35 years [4] and the majority of patients (90%) die before their 50s [2,4].
After repair, postoperative persistent arterial hypertension occurs in 7% to 33% of patients and may be a result of recurrent or residual CoA, or may be idiopathic. The frequency of postoperative recurrent CoA ranges from 5% to 50%. The significant variation in recurrence frequency is primarily associated with the age at initial repair [9, 10] rather than factors such as the specific surgical method of repair, suture material and sewing technique, which were once suggested to be associated with recurrence development [4].