CONCLUSIONS:
RV systolic dysfunction is a frequent finding in patients referred for
mitral valve surgery and is both a direct and indirect consequence of LV
volume overload. Tendyne TMVI resulted in an immediate improvement in RV
function, most likely due to abolition of MR with an associated
reduction in LV volumes and reduction in PASP. The preservation of
circumferential and radial strain despite an abrupt reduction in LV
volume loading, raises the possibility that the apical tether
redistributes LV contractile forces and enhances the interventricular
septal contribution to RV function. Further longitudinal studies of
larger cohorts are needed to confirm and extend our results.