Hemodynamic data:
Resting PASP measured invasively immediately before TMVI was elevated (≥25mmHg) in 29 patients (88%) (PASP 48±16mmHg; PADP 26±8mmHg; MPAP 34±8mmHg). Although there was no change in PA pressures immediately after TMVI (PASP 48±13mmHg; PADP 27±7mmHg; MPAP 35±9mmHg; p=0.18) (Table 4), there were immediate increases in contractility as evidenced by an increase in RVSWI (from 7±4g/m/beat/m² to 11±5 g/m/beat/m², p<0.001) (Figure 3a), SVI (from 29±8 mL/beat/m² to 34±11mL/beat/m², p<0.001) and CO (from 3.7±1.1L/min to 4.6±1.4L/min, p<0.001) (Table 4). CVP and HR remained steady (Table 4).