4 | conclusion
Cardiac changes in patients with hydrogen sulfide poisoning are delayed and abrupt, which is difficult to maintain by conservative treatments. However , returning to normal cardiac function is still possible when providing strong cardiopulmonary support like IABP and VA-ECMO during the transition.
REFERENCES
1. Haouzia P, Sonobea T, Judenherc-Haouzib A. Hydrogen sulfide intoxication induced brain injury and methylene blue. Neurobiology of Disease. 2020;133:104474.
2. Maebashi K, Iwadate K, Sakai K , et al. Forensic Science International. 2011;207: 91–95.
3. Sonobe T, Haouzi P. Sulfide Intoxication-Induced Circulatory Failure is Mediated by a Depression in Cardiac Contractility. Cardiovasc Toxicol. 2016;16:67–78.
4. Ago M, Ago K, Ogata M. Two fatalities by hydrogen sulfide poisoning: Variation of pathological and toxicological findings. Legal Medicine. 2008;10:148–152.
5. Mo WC, Shen J, Huang XC, et al. Acute myocardial injury following hydrogen sulfide poisoning. Toxicology and Industrial Health. 2020.
FIGURE LEGEND
FIGURE 1 A and D, The electrocardiogram of them were both normal; B showed extensive elevation of ST-T in all the leads and ST-T of C was seen to be elevated in lead I, II, III, aVR, aVF and V3, V4, V5, and V6.
FIGURE 2 A, Myocardial biopsy showed cardiomyocyte hypertrophy; B, Disorders of myocardial fibers, tissue sarcolysis and hypertrophy, irregular nuclei as well as swelling and dissolved mitochondria (arrows) were observed under electron microscope.
FIGURE 3 EF showed in the echocardiography returned to normal. LV, left ventricle; LA, left atrium; RA, right atrium.
FIGURE4 Cardiac contrast-enhanced magnetic resonance images showed nothing wrong with its perfusion and signal.
Movie S1. Reduced diffuse cardiac activity could be seen in the echocardiography. Its video coder unit was MPEG-4.