Introduction
Coronavirus (COVID-19) infection exposes patients with heart failure to a higher risk of morbidity and mortality.[1] Asymptomatic or mildly symptomatic COVID-19 respiratory virus infections can include fever, coughing, tiredness, dyspnea, diarrhea, headaches, and myalgia. Respiratory rate >30 bpm, arterial oxygen saturation less than 93% at rest, development of acute respiratory distress syndrome, septic shock, metabolic acidosis, and coagulopathy, including disseminated intravascular coagulation, are characteristics of severe cases. [2] In addition, patients infected with the COVID-19 virus have an increased risk of developing both venous and arterial thrombosis [3-6]
Thrombocytopenia and an increase in D-dimer are the hemostatic changes caused by COVID-19 that are most frequently observed, and they are linked to a greater need for mechanical ventilation, admission to intensive care, and death. It has been noticed that patients who are older and have comorbid conditions are more likely to have a high mortality rate, [7] which is believed to be greatly influenced by blood vessel dysfunction and clot formation, as indicated by elevated D-dimer levels brought on by blood clots.[8] Despite the that VAD thrombosis is a common complication, LVAD thrombosis due to COVID -19 is underreported and few cases are presented in the literature. Here we present a case of accelerated LVAD outflow thrombosis in the setting of COVID-19 infection with multiorgan failure.