Hypotheses Testing by Axis.
Given that we do not expect to see relationships with stress in the Placebo group, all following models will be applied on the experimental group, only (N ≈ 100).
Hypothesis 1: Executive Functioning and Stress Phases. We will run multilevel models for each stress outcome (STAI, heart rate, HRV, electrodermal activity, and blood pressure), with executive functioning facets, time (baseline, instructions, MAST, post-MAST) and the interaction terms between executive functioning and time as predictors, along with relevant covariates that correlate with the dependent and independent variables. We will perform correlations between all independent variables to optimize model parsimony.
Hypotheses 2, 3, and 4: Mediating roles of Cognitive Appraisals, Negative Repetitive Thinking, and Emotion Regulation. In an exploratory manner, we will run partial correlations (Pearson or Spearman depending on the normality of the distribution) between the executive functioning facets and all mediators. We will run three types of multiple mediation models using bootstrapping with Haye’s PROCESS macro in SPSS Statistics (IBM Corp., 2017), with cognitive appraisals (four mediators), repetitive negative thinking (two mediators), and emotion regulation (two mediators). For parsimony reasons, we will conduct these models when we observe a significant correlation between (a) an executive functioning facet and one potential mediator, and (b) one mediator and a stress outcome. In addition, we will include baseline stress levels for all reactivity models, and the reactivity stress levels as covariates for the recovery models. This will capture change from baseline to reactivity, and from reactivity to recovery. We will report p -values and consider results as significant when they fulfill the criterion of p < .05.
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