Corrosive esophagitis is characterized by caustic injury due to the ingestion of chemical agents, mainly alkaline substances such as detergents, cleaning compounds, and bleaches[1]. The severity of the injuries caused by caustic ingestion depends on gastric contents and caustic agent properties, including pH, concentration, ingested volume, and tissue contact duration[2]. In general, tissues are more severely damaged by alkaline solutions than by acidic solutions. This is because alkalis cause liquefactive necrosis, while acids cause coagulative necrosis resulting in eschar formation, thereby protecting the mucosal epithelium from deep ulcer formation. High-degree corrosive esophagitis may result in esophageal bleeding, perforation, and even death in the worst cases. In particular, esophageal strictures are one of the more serious complications of corrosive esophagitis. corrosive strictures are frequently very tight, long, eccentric, and multiple, and may require recurrent endoscopic therapy procedures, which are highly expensive. There is a belief that these patients have a very high stricture recurrence rate because of extensive esophageal damage and fibrosis. For these reasons, patients with corrosive strictures often undergo surgery In the past, which carries significant morbidity and mortality. But now, early endoscopic therapy is very useful in cases of corrosive esophageal strictures(CES), not only for determining the degree of mucosal injury and severity but also for predicting prognosis[3].