ACKNOWLEDGEMENTS
Dr. Takashi Sakurai is gratefully acknowledged for his generous gift of
HEK293 cells stably and inducibly expressing mutant and WT forms of RyR2
and co-expressing R-CEPIA1er, which were instrumental in the successful
completion of this work. The authors thank for technical and human
support provided by Mª Carmen Sampedro from SGIker of UPV/EHU and
European funding (ERDF and ESF), and by Neus Hernández, Mónica Espejo
and Jessica Jaramillo from the UAB for their excellent technical
support.