Fig. 6: Spatially controlled tßh expression in tßhnM18 mutant background.
Ubiquitous (actin), pan-neuronal (nSyb) and non-neuronal TDC (Tdc1) drivers significantly increase sugar responsiveness. Neuronal TDC (tdc2) and OA (NP7088) specific drivers do not alter sugar responsiveness. Boxplots depict total number of proboscis extensions in hemizygous mutant males with or without a UAS-tßh construct, and heterozygous for the Gal4 driver. Numbers indicate sample sizes, asterisks denote significant difference between the mutant its respective rescue group (Wilcoxon rank sum test, p < 0.05).